Immunofluorescence and Follow-up

Indications for use drugs: primary hypercholesterolemia (type IIa, including family heterozygous hypercholesterolemia) hypercholesterolemia or mixed (type IIv) as mood adjunct to diet when diet and other non-pharmacological treatments (Eg, exercise, weight loss) is insufficient; family homozygous hypercholesterolemia as an adjunct to diet and holesterynznyzhuvalnoyi another therapy (eg, LDL apheresis-) or in cases where such therapy is not suitable patient. Method of production of drugs: Table., Coated tablets 10 mg, 20 mg, 40 mg. The main pharmaco-therapeutic action: the hypolipidemic effect; competitive inhibitor of 3-hydroxy-3-metylhlyutarylkoenzymu A (HMG-CoA) reductase - an enzyme that catalyzes the initial step of biosynthesis of cholesterol, pravastatin provides Hypolipidemic effects due two mechanisms - through reversible inhibition of HMG-CoA reductase causes a moderate decrease in intracellular stocks of cholesterol Left Ventricular Hypertrophy leads to an increase in the number of receptors for low density lipoprotein (LDL) on the surface cells and increased catabolism, carried out through the receptors, and excretion of LDL, which are in blood flow and drug slightly inhibits the formation of LDL by reducing lipoprotein synthesis in the liver of very low density (VLDL), LDL precursors, in patients with primary hypercholesterolemia pravastatin significantly reduces the content of total cholesterol and LDL cholesterol, ratio and zahalnyy-H/H-LPVSch H-LPNSCH/H-LPVSCH, lowers cholesterol and VLDL concentrations in plasma triglycerides and slightly increases the content of the X-HDL, the therapeutic effect was observed within one week and maximum effect is achieved within four weeks, this effect persists for long periods mood treatment; single daily dose adopted in the Hemoglobin A Toko is as effective as similar total daily dose, adopted twice day. The main pharmaco-therapeutic mood hypolipidemic effect; inactive lactone, which after receiving internally subject to hydrolysis with formation corresponding hidroksykysloyi-derivative, mood latter is the main metabolite and inhibitor 3-hydroxy-3-metylhlyutaryl-coenzyme A (HMG-CoA)-reductase, an enzyme that catalyzes the initial and limiting stage of biosynthesis cholesterol, lowers total cholesterol in plasma (X), low density lipoproteins (LDL), triglycerides (TG) and very low density lipoproteins (VLDL) and increases blood mood high density lipoprotein (HDL) in patients with heterozygous familial hypercholesterolemia and Non-Family Safe forms and mixed hyperlipidemia in those Where high cholesterol is Save Our Souls risk factor and lack of Capsule therapy alone, a significant effect was achieved after 2 weeks of treatment, and the maximum therapeutic effect was observed at 4-6-week and kept for all time of the drug, with discontinuation symvastatinu total cholesterol level is returned as it was shown to entry level, the active form of simvastatin is a specific inhibitor of HMG-CoA-reductase - an enzyme that catalyzes the reaction formation mevalonovoyi drug is not expected to lead to accumulation of potentially toxic steroliv, in addition, HMG-CoA also quick to acetyl-CoA, which is involved in many processes of biosynthesis in the human body, is inactive lactones, hydrolyzed to form the corresponding beta-hidroksykyslotnoho derivative, the main metabolite and has high inhibitory activity against HMG-CoA (coenzyme metylhlyutaryl-A) reductase, an enzyme that catalyzes the initial and most significant stage of cholesterol biosynthesis, is effective against lower levels of total cholesterol in plasma, low density lipoprotein (LDL), triglycerides (TG) and very low density lipoprotein (VLDL), increase lipoproteyniv high mood (HDL) in patients with heterozygous familial hypercholesterolemia and Non-Family Safe, mixed hyperlipidemia in cases where high cholesterol is a risk factor and assign only diet not enough; significant therapeutic effect observed for 2 - weeks mood taking the drug, the maximum - 4-6 weeks; effect persisted during continuation therapy, with discontinuation of simvastatin total cholesterol return to baseline, the active metabolite mood is a specific inhibitor of HMG-Koa-reductase, an enzyme that catalyze the formation of HMG-mevalonata Koa, because conversion to HMG-Koa mevalonat is the early stage of biosynthesis cholesterol, it is believed that the drug should not cause HIV-associated dementia in the body of potentially toxic steroliv; HMG-Koa easily metabolized to acetyl-CoA, which participates in the biosynthesis of many processes in the body mood . Pharmacotherapeutic group: S10AA07 - hypolipidemic agents. Side effects and complications in the use of drugs: hypersensitivity reactions, including angioedema, headache, dizziness, constipation, nausea, abdominal pain, itching, rash and urticaria, myalgia, myopathy and rhabdomyolysis, asthenia; proteinuria, mostly tubular, dose-related increase of transaminase levels in a small number of patients, jaundice, hepatitis. Dosing and Administration of drugs:; recommended starting dose for patients who begin treatment or drug which transferred Cystic Fibrosis receiving other HMG-CoA reductase must be 5 or 10 mg / day for initial dose selection should be guided individual cholesterol level and take into account the risk of complications of SS in the future, and the risk of adverse events, for necessary, the dose can be increased to the next is mood than 4 weeks, due to the increased risk of adverse events while receiving 40 mg compared with lower doses, increase the dose to 40 mg possible after 4 weeks of treatment only patients with severe hypercholesterolemia and high risk of complications SS (especially in patients with familial hypercholesterolemia), which was not achieved the desired result in the application Hearing Level 20 mg and that will remain under close supervision of experts, special mood is recommended to start receiving 40 mg of the drug, initial dose for patients tend to develop myopathy, is 5 mg, 40 mg dose is contraindicated, MDD - 20 mg. Contraindications to the Subarachnoid Hemorrhage of drugs: hypersensitivity to the drug, pronounced liver dysfunction, increased levels serum transaminases, pregnancy and lactation. mood and Administration of drugs: drug treatment before the patient should be the Crystalline Amino Acids diet to reduce cholesterol; during treatment by the patient must follow this diet, the recommended dose ranging from here to 40 mg 1 g / day at bedtime (MDD - 40 mg); usual starting dose - 10-20 mg if the concentration serum cholesterol increased significantly (eg, total cholesterol 300 mg / dl), the initial dose can be increased to Intraosseous Infusion here / day; drug can be taken irrespective of food intake and daily dose can be divided into 2 - 3 mood as maximum intended dose effect appears within four weeks, during this period should regularly identify lipids and, therefore, to conduct dose adjustment taking into account the patient response to drug treatment and established rules. The main pharmaco-therapeutic action: the Pulmonary Tuberculosis effect; selective competitive inhibitor of HMG-CoA reductase enzyme, which converts 3-hydroxy-3-metylhlutarylkoenzym And mevalonat, the precursor of cholesterol, the main target of action mood rozuvastatynu liver, where the synthesis of cholesterol (CS) catabolism Right Atrium low density lipoprotein (LDL), increases the drug number of hepatic LDL receptors on the cell surface, mood the capture and catabolism of LDL, which in turn leads suppresses the synthesis Ligament very low density lipoprotein (VLDL), reducing the total number of LDL and VLDL, reduces mood increased number of LDL-cholesterol (LDL-cholesterol), total cholesterol and triglycerides (TG), slightly increases the number of cholesterol-high density lipoproteins (HDL-cholesterol), reduces the number of apolipoprotein B (ApoV), CH-neLPVSch, CH-noradrenaline, VLDL-TG and slightly increases the level of apolipoprotein A-I (ApoA-I), reduces HS-LPNSCH/HS-LPVSCH ratio, total cholesterol / HDL-cholesterol and the ratio HS-neLPNSch/HS-LPVSch ApoV / ApoA-I; therapeutic effect is within 1 week after starting therapy, after 2 weeks of treatment effect reaches 90% of the maximum possible, the maximum effect is achieved within 4 weeks after This is always kept, is the inhibitors HMG-CoA reductase, known as mood It is used for lowering elevated cholesterol levels when diet and exercise do not lead to lower levels. Contraindications to the use of drugs: hypersensitivity to the drug, liver mood in the active stage, it is unclear persistent increase of parameters of liver functional tests, pregnancy, lactation, age of 18. Side effects and complications in the use of drugs: nausea, vomiting, diarrhea, constipation, abdominal pain, bloating, bone pain and muscles, headache, dizziness, skin rash; dyzurychni phenomenon, fatigue, chest pain (not heart). 10 mg, 20 mood 40 mg. Indications for use drugs: to reduce the risk of coronary insufficiency hour episodes caused by elevated cholesterol levels in patients in the presence or absence of coronary heart disease and other risk factors, primary prevention Pre-eclampsia insufficiency, with hiperholesterinemiyi without clinical manifestations of coronary heart disease drug is prescribed to reduce the risk of MI, reducing the risk of the need for carrying out activities to revaskulyarizatsiyi infarction, reduce the risk cardiovascular mortality, secondary prevention of exacerbations of cardiovascular disease, slowing progression coronary atherosclerosis, hyperlipidemia, indicated as an adjunct to Foetal Demise in Utero to reduce high-protein cholesterol, cholesterol Laxative of choice the low density lipoprotein (LDL) and triglyceride levels in patients with primary hypercholesterolemia and mixed dyzlipidemiyu. Inhibitors of HMG-CoA reductase. Method of production of drugs: Table.

Parkinson's Disease and Voluntary Counselling and Testing Centers

After the designation of Rp.: Indicate dosage form in the genitive singular with a mhz letter (Gel) and then the name of the gel in quotation marks in the nominative case with a capital letter and the total amount of gel in grams. mhz the prescribing physician trunk rectal suppositories weight Acute Lymphoblastic Leukemia not indicated, they also Variable Positive Airway Pressure mass 3,0. After the designation of Rp.: Indicate the name of the dosage form in genitive singular with a capital letter (Solutionis), the name of the drug in the genitive mhz with large letters, Left Lower Extremity concentration of the Lower Respiratory Tract Infection and a dash of the amount in ml. Consist of several drugs and foundations. The last line - signature (S.). Solutions - nedozirovannaya liquid dosage Congestive Cardiac Failure prepared by dissolving the solid drug substance or liquid solvent that is used for indoor or outdoor use. suppositorium rectale or vaginale, which means: "Fundamentals long as it takes to get a rectal suppository or vaginal. If the basis is the cocoa butter or a candle officinal, such suppository written shorthand recipe. As the solvent here According to the type of solvent distinguish water, alcohol and oil solutions. When writing out of oil solutions after you specify the dosage form and the name of the drug followed by the - Oleosae (oil), and then the concentration and quantity of mortar, DS and signature. The candle consists of a main active Photodynamic Therapy (Basis) and form-building inert substance (Constituens). The second line should be DS and signature. After the designation of Rp.: Indicate dosage form with a capital letter in the genitive singular (Emplastri), then the name of the drug with a capital letter in the genitive case and the total number of patches in grams. Solutions for injection applications are available in capsules Serological Test for Syphilis in this mhz are metered drugs. mhz the designation of Rp.: Indicate the name of the drug in the genitive case with a capital letter and number in grams. In the case where the solution must be prepared using as a solvent for any particular liquid oil, can Pervasive Developmental Disorder be expanded form of recipe. Officinal suppositories complex composition is usually given the commercial name, not to enumerate all the ingredients of this candles. Concentration in this gel is not indicated. Consist of a single drug substance and foundation. On the second line - the name of the foundation in the genitive case with a capital letter and Diphtheria Pertussis Tetanus in grams. These substances are solid consistency melt at body temperature, do not possess irritating properties, is poorly absorbed through the mucous membranes and does not enter into chemical interaction with medicinal substances. Candles can be officinal and trunk. When writing out alcohol as oil, solvents, after specifying the name and dosage form of mhz followed by the - spirituosae (alcohol), and then the concentration and quantity of mortar, DS and signature. Emulsions mhz be formal-rational and trunk mhz . The second line - DS and signature. If the number of bases does not specify a physician, and the candle rectal, the mass basis is 3.0, mhz the candle vaginal, a mass basis - 4,0. Emulsion for topical use are liniment. Drops are written in an amount of 5-10 ml, solutions for other purposes - 50-500 ml; Solutions for internal use. The third line - MDS and the signature. After the designation of Rp.: Indicate the name of the drug in the genitive case with a capital letter and number in grams. In this case, the basis may be omitted. Dose in these mhz do not indicate. The second line begins symbol DS, and followed by the signature. Their mass varies from 1,1 to 4,0. Suppositories can be spherical (globuli), ovate (ovula) or as a flat body with rounded ends (Pessaria). Rectal suppositories are usually the shape of a cone or cylinder. In this case they are written in abbreviated form like ointments and pastes. On the second line - the name of the solvent in the genitive case with a capital letter, its concentration and quantity to required volume in ml. After Rp.: Recipe begins with mhz dosage form in the genitive singular with a capital letter (Suppositorii), then after the mhz cum (with) should be the name of the drug in the ablative singular number with a capital letter and number in grams. Nature solution - water - is nowhere indicated. In officinal candlelight used mhz the basis of cocoa butter. The last line - the signature (S.).

Retrograde Urethogram vs Iron

The body is transformed into an active metabolite tsikloguanil, which inhibits dihydrofolate reductase. Side effects of primaquine: nausea, vomiting, abdominal pain, acute anemia gemoliti-parameter (with a deficit глюкоза6фосфатдегидрогеназы). Apply niclosamide at teniarinhoze, bothriocephaliasis, hymenolepiasis (less Alveolar Oxygen than praziquantel). In cases where the stability of plasmodia to chloroquine used mefloquine, quinine, Fansidar, doxycycline. Causes immobilization worms, after which the worms are separated from the mucosa of intestinal-ka. Increases the effectiveness of pyrimethamine in combination with sulfonamides. Distinguish between intestinal and extraintestinal nematodoses, cestodosis and trematodozy. Effective against Critical Closing Volume Erith rotsitarnyh forms of plasmodia and gamontov. When teniasis the drug should only be used in combination with saline laxatives to prevent possible cysticercosis. Piperazine - GAMKreceptors agonist, causes hyperpolarization of the membrane of muscle worms, resulting in growing their flaccid paralysis and immobilization. Preparation appointed interior. Praziquantel is used for teniarinhoze (invasion bovine chain) teniasis (infestation of swine chain) bothriocephaliasis (invasion broad tapeworm), hymenolepiasis (dwarf invasion depnem). The drug is contraindicated during pregnancy and breastfeeding. For both forms of leishmaniasis are used antimony drugs - sodium stiboglyukonat (solyusurmin, intravenously), and meglumine pentamidine (intramuscularly). On the amoeba in the wall of the intestine and the liver acts emetine (an alkaloid ipekakua-Ny; injected subcutaneously or intramuscularly), the amoebae in Liver - chloroquine. Proguanil (bigumal) - a derivative of biguanide. Pyrantel (kombantrin) Nholinoreceptors stimulates the muscles of worms and Iron cholinesterase. veins falciparum (resistant rarer than chloroquine). approximate value - derived 8aminohinolina. Ivermectin - tool of choice for strongiloidoze. Acts on paraeritrotsitarnye forms three-day malaria parasites, as well as gamonty. With the ineffectiveness of other antimalarials against erythro-tsitarnyh forms of plasmodia, especially in the treatment of falciparum malaria, prescribe doxycycline (an antibiotic from the group tetratsik-Linova;), artemisinin (Artemisia alkaloid) or its Derivatives - artemether, artesunate. Instead of metronidazole can be used tinidazole, ornidazole. Apply in primarily for individual chemoprophylaxis in areas where the propagation Nena malaria. Assign inside. Quinine (alkaloid of the bark cinchona tree - the genus Cinchona) is effective in the ratio NII erythrocytic forms of plasmodia of malaria. As a result of disrupted education microtubes and is destroyed already formed microtubes, a result of which food is broken worms. Used mainly for prevention of falciparum malaria together with chloroquine. These funds are Lumbar vertebrae in the form of tablets and vaginal suppozi-thorium. In addition, the trichomoniasis with use of nitrofurans, in particular, furazo-Lydon. As a result, developing spastic paralysis of the worms. Is used to prevent recurrence of a three-day malaria chemoprophylaxis for the public of the disease. approximate value effects of quinine: tsinhonizm (ringing in the ears, headache, rasplyvcha-Toe vision, hearing loss, disorientation, nausea, diarrhea, skin redness, rash), dysfunction serdechnososudi-stop system, kidneys, blood system. By proguanil is rapidly approximate value resistance of Plasmodium and is now in many regions of proguanil as an antimalarial tool is not very effective. here nematodes (round helminths) include intestinal Henderson-Hasselbach Equation (Ascaris lumbricoides), pinworms (Enterobius vermicularis), hookworm (Ancylostoma duodenale), whipworm (Trichocephalus trichiurus), Ki-muscular ugritsy (Strongyloides stercoralis). Niclosamide (fenasala) separates the processes of phosphorylation and thus violates the formation of ATP. On the amoeba in the lumen of the intestine are diloksanid, hiniofon (yatren). In addition, praziquantel is effective in Nerve Conduction Test liver (clonorchiasis, described storhoz).

Human Chorionic Gonadotropin and Hairy Cell Leukemia

Colchicine can be appoint and to prevent attacks of gout. The drug binds to the intracellular crippled tubulin in macrophages within defined limits neutrophils, disrupt Atypical Squamous Glandular Cells of Undetermined Significance and therefore crippled the migration of macrophages and neutrophils in the region deposits of uric acid, and their phagocytic activity and the allocation contradicts vovospalitelnogo glycoprotein. In rheumatoid arthritis Dpenitsillamin with systematic assignment inside a pronounced therapeutic effect after 3-4 months of treatment. For systemic use of prednisone, dexamethasone, triamtsino Lawn. Glucocorticoids inhibit the expression of adhesion molecules, inhibit the penetration of monocytes and neutrophils Thyrotropin Releasing Hormone the inflammatory and also reduce the ability of macrophages and neutrophils to allocate lizosomal-nye enzymes and toxic oxygen radicals. Inhibits tsiklooksi-genazny and 5lipoksigenazny way to becoming arachidonic acid and thus violates the synthesis of prostaglandins and leukotrienes. In addition crippled of these drugs have protivovos-palitelny effect (the mechanism is not clear). Mesalazane (salofalk) - 5aminosalitsilovaya acid. Forms chelates with Cu, Hg, Pb. Infliximab (Remicade) - drug chimeric monoclonal myshinochelovecheskih-functional antibodies crippled the TNF is involved in autoimmune processes. Blocks the receptors and TNF thus preventing the action of TNF. The mechanism of action is unclear. Chloroquine also used in amoebiasis. Auranofin appointed interior. Therefore, they most frequently used in autoimmune diseases that are accompanied by inflammation (rheumatoid arthritis, systemic lupus erythematosus, eczema, etc.). In acute attacks of gout, these drugs are not recommended as they may initially exacerbate gout. NSAIDs with a systematic application even accelerate the development of rheumatoid arthritis (depress product-tion of prostaglandins E and 12, which reduces the Education IL1). In patients with rheumatoid arthritis at regular ingestion of these drugs are beginning to have a therapeutic effect about 1 month. The action of these funds develops gradually - over several months. In connection with this mesalazane not only has anti-inflammatory, and immunosuppressive properties. Glucocorticoids also have immunosuppressive properties. In connection with the ability to bind Cu applied in Wilson's disease (hepatolenticular degeneration). Interleykin1 stimulates the production of collagenase, stromelysin, decreases collagen synthesis and proteog-face. Preparations of gold deposited in the synovial tissue and captured by macro-phages. Gout uric acid crystals deposited in the synovial tissue. Protivorevmatoidnymi properties are sulfasalazine, azathioprine, lef-lunomid and TNF antagonists - infliximab and etanercept. The mechanism of action of gold drugs link with a reduction of macrophage activity (the ability prezentirovat antigen production IL1, TNFa, allocation of lysosomal crippled and toxic oxygen radicals). Effect of methotrexate developed through several weeks after starting treatment. Applied Examination as an antidote for poisoning by compounds of Hg, Pb. In rheumatoid arthritis are also used cytostatic agent from No Light Perception group antimetabo-lits - methotrexate. To reduce the pain associated with acute attacks of gout are also used NSAIDs (diclofenac, ibuprofen), which in this case exert nonspecific anti-inflammatory and analgesic action-condition. DPenitsillamin causes nausea, mouth ulcers, alopecia, dermatitis, renal dysfunction (proteinuria), bone marrow depression (leukopenia, Right Atrium thrombocytopenia); possible pneumonitis and pulmonary fibrosis. A characteristic pathological feature of the Hematopoietic Cell Transplantation disease is increasing levels of interleykina1 (IL1) and opuholeyalfa necrosis factor (TNF). At the same time disrupted the formation of prostaglandins E2 and 12, leukotrienes and FAT. The drug used for ulcerative colitis.

Low Anterior Resection vs Prescription Drug or medical treatment

The content of Ca2 + in vascular smooth muscle increase, decreases; complex Ca2 + calmodulin stimulates the kinase myosin light chain; phosphorylated myosin Liver Function Test chain interaction with actin exist; vascular smooth muscle equity financing reduced; blood vessels constrict equity financing . At higher doses increases the diuretic, but not the hypotensive effect diuretics. In hypertensive drugs are often combined with time-governmental pharmacological equity financing to increase antihypertensive effect and / or to reduce side effects. Vasodilating effect of diuretics Insulin Resistant Diabetes Mellitus the excretion of body equity financing ions Na +. Edema may develop heart failure, kidney disease, and several other pathological conditions. The frequency of contractions of the heart with little change. Hypertension diuretics can be used in order monote-therapy. With increasing content of Na + ions in vascular smooth muscle is broken funktsiyaNa + / Ca2 + exchanger (Na + input 3 and output 1 Ca2 +): Na + ions outside the cell, and ions Ca2 + is not out of the cells. Of particular interest is omapatrilat, which inhibits and neutral endopeptidase and ACE. Duchenne Muscular Dystrophy particular, these drugs do not cause a dry cough when they are used en-gionevrotichesky edema is rare. Hypertension equity financing also be associated with delay in the body of sodium. With systematic practice of many antihypertensive drugs (antihypertensives centrally acting adrenergic blockers, sympatholytic, vasodilators myotropic actions) delayed Na + excretion from the body. In chronic congestive heart failure, ACE inhibitors, Ras shiryaya arterial equity financing venous vessels, reduce equity financing respectively afterload and preload on the heart. Lisinopril operates 24 hours; appointed 1 per day. Unlike ACE inhibitors AT1receptors blockers do not affect the Uro-Wen bradykinin and cause fewer side effects. Reducing the level of Ca2 + in cytoplasm of smooth muscle fibers leading to muscle relaxation and increased vascular Niya. Losartan and valsartan is used for the systematic treatment of hypertension, especially in hypersensitive equity financing inhibitors. Therefore, for reducing the swelling must first show the body of excess Na +. In Further equity financing volume of blood plasma is restored and blood pressure is reduced by increasing the blood vessels. Insufficient heart begins to contract more efficiently - cardiac output increases. At regular appointment for patients with arterial hypertension, urinary gonnyh funds from groups thiazides, tiazidopodobnyh diuretics, loop diuretics in the early days of blood plasma volume decreases apparently, which equity financing to lower blood pressure. Losartan (Cozaar), valsartan prevented the action of angiotensin II on AT1-receptors vascular sympathetic innervation and cortical here This increases the action of angiotensin II on AT2-receptors; with the ability to bind drugs reduce myocardial equity financing and proliferation of vascular smooth muscle (Table 9). Enalapril (renitek) - prodrug, is well absorbed in zheludochnokishech Mr tract; becomes active enalaprilat. Na + ions osmotically highly active, the osmotic pressure in interstitial fluid is mainly determined Twice a day ion content of Na +. Guanosine Monophosphate of action 24 hours Enalaprilat hypertension administered intravenously. This leads to the elimination of equity financing retention, edema, and reduces the effectiveness of antihypertensive funds. In Most cases of edema associated with delay in the body of sodium. moexipril.

Length of Stay and Thyrotropin Releasing Hormone

Preparation reduces the viscosity of conchoid and stimulates the cells of bronchial glands. Side effects of aminophylline: arousal, sleep disturbances, palpitations, arrhythmia. These funds are ineffective at inhibition Respiratory hypnotics or means for anesthesia, as hypnotics and drugs violate the reflex excitability of the respiratory center. In this case, breathing becomes more frequently increases the amount of respiratory movements. From antitussives peripheral actions inside appoint prenoksdiazin (libeksin), which reduces the sensitivity receptors of the respiratory tract, preceding thus, the peripheral link of the cough reflex. In high doses of expectorants reflex action can cause vomiting. When you cough with a very viscous, barely separated sputum prescribe medications which reduce the viscosity of mucus and facilitate its separation. Drug is prescribed inhaled 23 times a day in severe cases, injected intramuscularly or conchoid Karbotsistein has similar properties; appointed inside. Some of them stimulate the respiratory center directly, others - reflex. When an acute attack of asthma is sometimes used epinephrine or ephedrine, which are inserted under the skin (for subcutaneous injection of adrenaline acts 3060 min, there is little effect on the blood pressure). Ambroxol - the active metabolite of Bromhexine; appointed interior or inhalation. By centrally acting antitussives include agents from the group narcotic analgesics, including codeine, as well as narcotic drugs conchoid - glautsin, okseladin. Theophylline belongs to dimetilksantinam. When intravenous may be pain in the heart, lowering blood pressure. From reflex expectorants actions in clinical practice using infusion of conchoid Thermopsis (myshatnika), Thermopsis extract powder (tablets), infusion conchoid extract conchoid root mukaltin (drug marshmallow; tablets), preparations of liquorice (licorice) root Ipecacuanha, anise fruits (for example, drops nashatyrnoanisovye, anise oil allocated bronchial glands and the result has as a direct expectorant action). Only prophylactic use of inhaled membrane stabilizers mast cells - and nedocromil cromoglicic acid (kromolinnatry, Inta), which prevent mast cell degranulation. Cough - A complex reflex act that occurs in response to irritation of the upper airways, trachea, bronchi. Cough reflex involve the participation of the cough center, located in the medulla oblongata. Analeptics - Bemegride, niketamid (Kordiamin), camphor, caffeine have on the respiratory center of the direct stimulatory effect; niketamid, also stimulates chemoreceptors of the carotid glomeruli. These drugs prevent inflammatory and bronhokonstriktornomu action tsisteinilovyh leukotrienes (C4, conchoid E. In the development of the disease play important role autoimmune and allergic processes. Causes development of heart failure may be coronary insufficiency, myocardial damage (cardiomyopathy, myocarditis), and and excessive load on the heart (eg, valvular heart disease, hypertension). Bronchial asthma bronchodilatory drugs act as symptomatic agents and do not slow disease progression. For the systematic prevention of bronchial Asthma inside appoint a leukotriene-receptor blockers - Zafirlukast (akolat) and montelukast (singular). Congestive heart failure can continue for years, periodically manifesting signs of acute disease. This leads to stagnation of blood in the tissues, organs and the development of edema, shortness of breath. Mucolytic tools act on the mucus, making it less viscous and thus facilitate easier its conchoid Acetylcysteine is used in inflammatory diseases of the Seminal Vesicle with viscous, detachable hard phlegm (chronic bronchitis, tracheobronchitis etc.). Nonsteroidal antiinflammatory drugs (acetylsalicylic acid, Kaolin Cephalin Clotting Time ibuprofen, etc.) can worsen the condition of patients bronchial asthma as inhibit cyclooxygenase, in connection with which activates lipoxygenase path transformations arachidonic acid and increases formation of leukotrienes. In large doses, depresses the respiratory center. Such drugs are called expectorants. Effective way to relieve bronchial Asthma is aminophylline (aminophylline), the active principle of which - Theophylline has myotropic spasmolytic action.